How can you prevent cancer from developing?

1.Theories about cancer triggers

It is mainly influences that alter the genetic material, carcinogenic.

The cell is particularly sensitive to this during cell division; therefore, cells that divide quickly are particularly vulnerable.Influences that prevent the immune system from detecting and eliminating degenerate cells are also considered to promote cancer. Particularly dangerous are therefore:

Environmental toxins and physical radiation

• Physical noxes

  • Ionizing ‘tradiation’ like ultraviolet ‘tlight,’ x-ray-toder radioactive radiation.

For example, s.t. In the cardiac examination by means of computed tomography, patients are buying the increased sensitivity of the examination method with an increased risk of cancer.For example, American scientists calculated that in twenty-year-olds, one in 143 women who had been examined by coronary CT had cancer during their lifetime as a result of this ‘tangiography radiation’, but only one in 686 men of the same age. Coronary angiography seems to significantly increase the risk of cancer, especially in women and young people. [21 tAfter a skull CT, the risk of cancer for women is 1:8,100 and for men 1:11,080.[22 t-t-based CT scans performed in the United States in 2007 are associated with 1:8,000 future cancers.[22) 23

  • Chemical noxes
  • Mutagen chemicals.
  • The most important are larger polycyclic aromatic hydrocarbons, tBenzene, chromium(VI) compounds, and nitrosamines.

    Biological influences

    • Oncoviruses (estimated by the American Cancer Society about 17% of cancer cases[24)
    • Various DNA viruses, .tz.

    b.e. the hepatitis B virus (HBV), which can lead to liver cell carcinoma, hpV, tdas to cervical cancer

  • Various RNA viruses, .tz.
  • b.e. the hepatitis tC virus

  • Miscellaneous retroviruses, .tz.
  • B. HIV, stHTLV

  • Stem cells, especially embryonic ones, can cause cancer in certain circumstances. [25
  • Immunosuppressive therapies after an organ transplant can increase the risk of cancer to three-[26-tup-six-fold) compared to the normal population.[ 27 ‘tThe most common cancers after a transplant include the ‘tKaposi sarcoma’ and other forms of skin cancer, known as PTLD, a group of various lymphomatic diseases. [28 “TIn general, the immune system plays an important role in the development of cancer, because it is basically able to detect degenerate cells and successfully combat them. [29
  • Body size: Studies in both men and women show a link between body size and cancer risk in later life.
  • Only in animals: some ttumors are transferable from animal to animal, among the infectious ttumors belong, among other things, the sticker sarcoma of dogs and the DFTD (DevilFacial tTumourDisease) – the bag devil.
  • Lifestyle and living conditions

    • The Million Women Study confirmed the assumption that being overweight increases the risk of cancer.

    An increased body mass index increased both the incidence of cancers: endometrial cancer, esophageal cancer, trendocancer, tMultiples, tmyeloma, tpancreas carcinoma, tOvarian cancer, tmammacancer, and colon cancer after menopause. According to the study authors, 5% of all cancer cases can be reduced to obesity.

  • Psychic Causes -That the personality or certain internal conflicts cause cancer is unconfirmed.
  • It is conceivable at best that mentally stressed (e.g. stressed) people behave more riskily (e.g. smoking more).

    It is an art of lifestyle to avoid these causes.

    Cancer is also considered by some scientists to be an out-of-control healing process in which the body forms trophoblasts to overcome tissue damage or tissue aging.
    These cells are protected from white blood cell attack by an electrostatically charged protein shell (pericellular sialoculine envelope).

    However, if enough pancreatic enzymes act on them, the protective shell is broken down and the leukocytes (white blood cells) can eliminate the cells.Nature has thus given the pancreas the important task of controlling trophoblast cells and thus preventing cancer.

    Professor Beard, who developed the trophoblast theory of cancer development, suspected that there must be a nutritional factor in addition to the enzymes.

    It was fascinating that the Washoe Indians almost never suffered from respiratory diseases and that they used “Dortzawasser”, a brew from the root of a petersilien-like wild plant (Leptotaenia dissecta) as a preventive remedy.The seeds of the Prunus-rosacea family contain a lot of them: bitter almonds, apricots (seeds), blackthorn, cherries, nectarines, peaches, plums…. But they can also be found in grasses, corn, sorghum, millet, cassava (root nodules of the cassava plant), flaxseeds and apple kernels.

    Nitrilosides occur together with the other B vitamins and are also water soluble.Since it was the 17th such substance, a scientist named Cancer identified it as vitamin B17: “Since the nitrilosides are neither food nor medicines, they can be described as accompanying food components.”

    Anyone who owns a dog or cat has already observed that these pets often specifically seek out certain grasses to eat, even though they are quite saturated with other food.This happens especially often when the animals are not doing well. Interestingly, they instinctively choose the grass varieties with a particularly high content of vitamin B17.

    When monkeys get fresh peaches or apricots at the zoo, they carefully peel the flesh, crack the hard stone and greedily eat the soft core.In the past, people ate more foods containing nitrilosides, such as millet and apples with the seeds. Today’s refined foods barely contain this vitamin. As a result, the incidence of cancer has steadily increased.

    Vitamin B17 – also known as laetril – or amygdalin consists of two glucose units, benzaldehyde and cyanide, both of which form a stable compound.

    Cyanide is highly toxic and deadly in higher doses.However, in its naturally bound form, it is chemically inactive and has no effect on living tissue. Only one substance can break down Laetril, the enzyme beta-glucosidase.

    When Laetril meets this enzyme in the presence of water, both the cyanide and the benzaldehyde are released, which is also highly toxic in isolation.When the two substances work together, they are at least 100 times as toxic as any substance in itself (synergy effect).

    The cleft enzyme beta-glucosidase is present in the body exclusively in cancer cells in large quantities, often more than 100 times the concentration of healthy cells.Thus, vitamin B17 is broken down in the cancer cell and its toxins only work there.

    Another important enzyme called rhodanese (thiosulfate sulfur transferase) acts as a protective enzyme because it can neutralize cyanide and convert it into by-products that are useful for health.It is found in large quantities throughout the body, except in cancer cells. Healthy tissue is thus protected, as the protective enzyme completely neutralizes the effect of the fission enzyme. On the other hand, the cancer cell is very sensitive to the release of cyanide and benzaldehyde because it lacks the protective enzyme and has a much higher concentration of fission enzyme.

    The dual property of vitamin B17: Healthy tissue can use vitamin B17 as a nutrient while poisoning the cancer cell.

    There are many theories about cancer development and possible cancer cure.I chose them. Since I am not a physician, but have only been responsible for scientific research in my institute, I decided to use a compilation of the available literature and point out that it is not a substitute for medical treatment by a Specialist.

    Literature

    • H.-J. Schmoll, K. Höffken, K. Possinger (eds.): Compendium Internistic Oncology.4.Edition. Springer, Berlin 2005. 3 volumes. ISBN 978-3-54020657-6.

    limited preview in Google Book Search

    • Cancer in Germany, frequencies and trends.

    4th edition 2004, published by the Working Group on Population-Related Cancer Registry in Germany, Saarbrücken (= “Cancer Atlas”)

  • w.
  • U. Eckart (ed.): 100 years of organized cancer research; 100 Years of Organized Cancer ,Thieme, tStuttgart 2000.201 S. ISBN . . . . . . . . . . . . . . . . . . . . . . . . . . . . •restricted “tPreview” in googlebook search

  • Siddhartha tMukherjee: “TThe Emperor of All Maladies: A Biography of Cancer
  • 2010. •tISBN -t1-4391-0795-5. •restricted “tPreview” in google book search. German edition: The King of All Diseases: Cancer – a Biography, “tDuMont Buchverlag, Cologne 2012, ISBN “t978-3-83219644-8”.

  • h.
  • Stamatiadis-Smidt, H. Pcs. Wiestler (ed.): Theme of cancer.3.Edition. “tSpringer, Berlin 2006. ISBN .t3-540-25792-6. •restricted “tPreview” in Google Book Search

  • Bruce’tN.
  • Ames, s.t.S. Gold: False assumptions about the connections between pollution and the development of cancer. In: “Applied Chemistry.” 102, No. 11, 1990, pp. 1233-1246, étdoi:10.1002/an.19901021106.

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